Structure and Function of AmpC Beta-Lactamases

Presentation Type

Poster/Portfolio

Presenter Major(s)

Chemistry

Mentor Information

Brad Wallar

Department

Chemistry

Location

Henry Hall Atrium 99

Start Date

11-4-2012 9:00 AM

Keywords

Life Science

Abstract

Beta-lactamases are enzymes that are produced by bacteria that hydrolyze the beta-lactam ring in many different types of antibiotics (such as amoxicillin, cephalosporin, etc.) and thus play an important role in antibiotic resistance. These clinical applications are primarily focused on determining how to inhibit this hydrolysis and ultimately sustain the antibiotic well enough for it to complete its intended use. The research project in question has been focusing on AmpC, which is a class C beta-lactamase that can inactivate a range of known antibiotics. AmpC has been the focus for research due to the fact that, in a similar class C enzyme, P99, a mutant at the asparagine-152 residue has been found to completely change the profile of antibiotics that can bind in the active site. Using a combination of site-directed mutagenesis, protein purification, enzyme kinetics, and x-ray crystallography, we have been developing a structure-function relationship for the asparagine-152 residue.

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Apr 11th, 9:00 AM

Structure and Function of AmpC Beta-Lactamases

Henry Hall Atrium 99

Beta-lactamases are enzymes that are produced by bacteria that hydrolyze the beta-lactam ring in many different types of antibiotics (such as amoxicillin, cephalosporin, etc.) and thus play an important role in antibiotic resistance. These clinical applications are primarily focused on determining how to inhibit this hydrolysis and ultimately sustain the antibiotic well enough for it to complete its intended use. The research project in question has been focusing on AmpC, which is a class C beta-lactamase that can inactivate a range of known antibiotics. AmpC has been the focus for research due to the fact that, in a similar class C enzyme, P99, a mutant at the asparagine-152 residue has been found to completely change the profile of antibiotics that can bind in the active site. Using a combination of site-directed mutagenesis, protein purification, enzyme kinetics, and x-ray crystallography, we have been developing a structure-function relationship for the asparagine-152 residue.