Characterizing Protein-protein Interactions for Accurate Cell Division in Fission Yeast

Presentation Type

Poster/Portfolio

Presenter Major(s)

Biomedical Sciences

Mentor Information

Dawn Clifford Hart

Department

Cell and Molecular Biology

Location

Kirkhof Center KC56

Start Date

10-4-2013 9:00 AM

End Date

10-4-2013 10:00 AM

Abstract

Cell division is a necessary process for growth and development. Fission yeast (S. pombe) cells provide a model system to study polarity and cytokinetic mechanisms because, like human cells, they grow in a bipolar fashion and divide symmetrically through contraction of an acto-myosin ring. Mid1 is a founding protein of the acto-myosin ring that helps recruit ring proteins and define the division plane. Without Mid1, there is incomplete, uneven division. The orb class of S. pombe mutants is classified by the loss of cell polarity and round shape. To study if these polarity defects are related to cytokinetic defects, we have examined interactions between Mid1 and orb mutants. These orb mutants show more binucleate cells with internal septa, paired configurations, differing localizations of Mid1, and higher protein levels of Mid1. These phenotypes suggest a relationship between cytokinetic defects and the polarity genes, as well as a link between Mid1 and the Orb proteins.

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Apr 10th, 9:00 AM Apr 10th, 10:00 AM

Characterizing Protein-protein Interactions for Accurate Cell Division in Fission Yeast

Kirkhof Center KC56

Cell division is a necessary process for growth and development. Fission yeast (S. pombe) cells provide a model system to study polarity and cytokinetic mechanisms because, like human cells, they grow in a bipolar fashion and divide symmetrically through contraction of an acto-myosin ring. Mid1 is a founding protein of the acto-myosin ring that helps recruit ring proteins and define the division plane. Without Mid1, there is incomplete, uneven division. The orb class of S. pombe mutants is classified by the loss of cell polarity and round shape. To study if these polarity defects are related to cytokinetic defects, we have examined interactions between Mid1 and orb mutants. These orb mutants show more binucleate cells with internal septa, paired configurations, differing localizations of Mid1, and higher protein levels of Mid1. These phenotypes suggest a relationship between cytokinetic defects and the polarity genes, as well as a link between Mid1 and the Orb proteins.